Bats in Laos Caves Contain Newly Found Closest Relative to SARS-CoV-2
Oddly, No Furin Cleavage Site on These Spike Proteins....hmmm.
Rhinolophus bats from the environs of Laos contain reservoirs of what scientists refer to as “a bunch” of coronaviruses closely related to SARS-CoV-2. Based on sequencing data, these are now the most closely related Rona virums known to science, even closer than the Rhinolophus affinis-derived strain called RaTG13 isolated around 2015. (There had been a giant question mark about what that strain had been doing in the years since the sample was first retrieved.)
None of these viruses has a furin (ubiquitous human protease) cleavage site in the stalk of the Spike (S) protein. The probability that such a sequence would “suddenly evolve” in bats then “jump” to humans as proposed by Fauci is ludicrous on it’s face.
Engineering furin-cleavage sequences into viruses is a common Gain of Function subject. Natural addition of this sort of sequence had been observed in some types of other viruses and was noted to be an infection-enhancement alteration, so it seemed like a cool idea to make a Frankenstein chimera containing this sequence that allows the enzyme to snip the S protein in half and solubilize it. Vaxx manufacturers copying the entire Chinese-provided S-protein code exactly and uncritically, including the man-made furin cleavage site, is perhaps the most damaging thing about mRNA vaccines to date.
What is interesting about this paper is that it carefully tries to avoid robust wave-making claims and, I assert, couches all findings in language that the average journo would easily miss as an indictment of The Narrative. Saying things in the Abstract like “bind as efficiently to the hACE2 protein as the SARS-CoV-2 Wuhan strain” then later saying in the meat:
A common feature of zoonoses (animal species-jumping virums) is that they typically are capable of back-jumping fairly easily. Got a swine flu suspect virum from within an infected human host? Slap it onto some pig cells and see what happens. Chances are it will easily re-infect the swine source of production of it’s literal grandparent virum. Take a standard non-infectious virum living in peaceful coexistence with swine, slap it on human cells, and the probability is that the virum will have no interest, no easy access mechanism.
This paper showed that the “BANAL” (the naming of this strain with a meaningful English word, I submit, is no accident) virums obtained from these Lao bats are basically uninterested in humans, incapable of making the jump. This, despite being very, very closely related to the Rona O.G. strain that we know has a taste for humans. The Lao virums have no furin cleavage site and the hACE2 receptor affinity of the S protein RBD (receptor binding domain) is simply not strong enough to easily make a pathogen. So what happened? I will point to China’s impeccable record on human rights and the kind manner in which they treat dissident and religious citizens to let the reader draw their own conclusions about what really happens in their Western Desert prisons.
If there are still gaps in understanding, ask why every other zoonotic event ever witnessed in all of medico-scientific history has resulted in rapid, turbocharged genetic sequence speciation (a dramatic uptick in mutation rate) upon arrival in a new host species. Yet, retained donor blood samples show a virum sequence present in the U.S. in March 2019 that remained remarkably stable and nearly sequence-identical with Chinese samples through their (denied) outbreak starting October 2019 through October 2020, and also remaining basically identical for Americans, Europeans and Indians through about November 2020 at which point, there was sudden speciation. It was as if a new host jump had happened, or new selective pressure had been applied to the virums. The temporal relationship between the advent of COVAXIN first, then widespread deployment of mRNA vaxxes a month later, followed by a worldwide variant outbreak is something only ideologues would ignore.
Furin
Several labs have undertaken to add furin cleavage sites, including Institut Pasteur (same one as where this paper’s authors are from, so some intrigue there), the Ralph Baric Lab of the University of North Carolina under some sort of relationship with Peter Daszak of exceedingly deceptive EcoHealth Alliance fame, and the National Microbiology Lab in Winnipeg where Chinese spies were allowed free reign, and of course, Wuhan Institute of Virology where the “Batlady” scientist Shi Zhengli was very forthcoming with genetic sequences that she helpfully provided to Baric upon request.